Dentinhypersensitivity (DH) and also termedsensitive dentin, dentin sensitivity and cervical hypersenstivityis described as a short, sharp pain as arising from exposed dentin in reactionto stimuli (evaporative, osmotic, thermal, chemical or tactile) which can notbe related to any other form of dental defect. It can also diagnosed as a dullor throbbing pain. Dentin and pulp have the sameorigin, where both are derived from dental papilla and considered as one organ,although dentin does not feel pain or sensitivity thepain is due to response of the pulp nerves to stimuli at the site of the dentinsurface.(1) Adegree of dentin sensitivity is normal, but pain is not usually experienced dailylike drinking a cold beverages.
There are theories explaining the mechanism ofdentin hypersensitivity, but the hydrodynamic theory is the most widelyaccepted. The hydrodynamic theory states that if dentin is subjected to a provocativestimulus, a movement of dentinal fluid within the tubules takes place, creatinga change in pressure inside the dentin and initiating a response in the pulpnerve ends, eventually causing pain for the patient.(2) Thedentin pulp complex might react to the insufficient protection by slowly depositioningtertiary dentin as a physiologic repair mechanism, thus increasing the insulatingbarrier between pulp and the stimulus thus, decreasing signs of hypersenstivity.(3) It is challenging to diagnose DH, Carried out byexclusion. Once reached, all other possible pain causes are ruled out, wherean detailed patient history and clinical examination are required.(4)The investigation includes a pain senstivity testby gentle air streaming from a dental instrument onto the sensitive area, orgentle scratching with a dental probe. Ifa negative result for the pain senstivity test is found, no management for hypersensitivityis required and further reason is expected, such as other causes of orofacial pain.
(5) Various treatment modalities have been recommended. Mostly, categorizedinto clinical managment performed by the specialist, this modalities act via either reducing the diameter of dentinal tubulesby using tooth pastes, varnishes, resins(6) or nerve fibers usingpotassiumchloride, or potassium nitrate potassium citrate to desensitizate/block neural transmission .(7) Other managments include the using of bonding agents and laserapplications.
(8) Other maneuvers that can be performed at home by brushing usingsoft tooth brushes and educating the patient the correct brushing technique avoidingover-brushing, using fluoridated mild abrasive toothpastes, and using potassiumsalts based rinses and sugarless gums. Sofar, no entirely recognized gold-standard management, which reliably cures thepain of dental sensitivity in the extended term and satisfy the dentist and thepatient, was found. Thereforeit seem to be of value to study the effect of photo polymerized versuschemical cured glass ionomer based desensitizing agents on patency of thedentinal tubules aiming to decrease the dental sensitivity.